M. L. Bland. 2022. Regulating metabolism to shape immune function: Lessons from Drosophila. Seminars in Cell & Developmental Biology. Available online April 16, 2022.
B. A. Martínez, Hoyle, R.G., Yeudall, S., Granade, M.E., Harris, T.E., Castle, J.D., Leitinger, N. and M.L. Bland. 2020. Innate immune signaling in Drosophila shifts anabolic lipid metabolism from triglyceride storage to phospholipid synthesis to support immune function. PLOS Genetics. 16 (11):e1009192.
M. Suzawa, Muhammad, N.M., Joseph, B.S. and M.L. Bland. 2019. The Toll signaling pathway targets the insulin-like peptide Dilp6 to inhibit growth in Drosophila. Cell Reports. 28 (6): 1439–1446.
S.W. Roth, M.D. Bitterman, Y.J. Lee, M.J. Birnbaum and M.L. Bland. 2018. Innate immune signaling in the Drosophila fat body blocks DILP signaling by uncoupling PI(3,4,5)P3 production and Akt activation. Cell Reports. 22 (10): 2550-2556.
Bland, M.L. 2016. Measurement of Carbon Dioxide Production from Radiolabeled Substrates in Drosophila melanogaster. Journal of Visual Experiments. Jun 27;(112). doi: 10.3791/54045.
Bland, M.L. and M.J. Birnbaum. 2011. Cell Biology. AdaPting to Energetic Stress. Science. 332 (6036): 1387-1388.
Bland, M.L., R.J. Lee, J.M. Magallanes, J.K. Foskett and M.J. Birnbaum. 2010. AMPK promotes growth in Drosophila melanogaster by regulating nutrient uptake and visceral muscle function in the gut. Developmental Biology. 344 (1): 293-303.
DiAngelo, J.R., M.L. Bland, S. Bambina, S. Cherry and M.J. Birnbaum. 2009. The immune response attenuates growth and nutrient storage in Drosophila melanogaster by reducing insulin signaling. Proc. Natl. Acad. Sci. 306 (49): 20853-20858.
Bland, M.L., R.C. Fowkes, and H.A. Ingraham. 2004. Differential requirements for SF-1 gene dosage in adrenal organogenesis versus adult endocrine function. Molecular Endocrinology. 18: 941-952.
Bland, M.L., C.A.M. Jamieson, S.F. Akana, S. R. Bornstein, G. Eisenhofer, M.F. Dallman, and H.A. Ingraham. 2000. Haploinsufficiency of steroidogenic factor-1 in mice disrupts adrenal development leading to an impaired stress response.
Proc. Natl. Acad. Sci. 97 (26): 14488-14493.