M. L. Bland. 2022. Regulating metabolism to shape immune function: Lessons from Drosophila. Seminars in Cell & Developmental Biology. Available online April 16, 2022.
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B. A. Martínez, Hoyle, R.G., Yeudall, S., Granade, M.E., Harris, T.E., Castle, J.D., Leitinger, N. and M.L. Bland. 2020. Innate immune signaling in Drosophila shifts anabolic lipid metabolism from triglyceride storage to phospholipid synthesis to support immune function. PLOS Genetics. 16 (11):e1009192.
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M. Suzawa, Muhammad, N.M., Joseph, B.S. and M.L. Bland. 2019. The Toll signaling pathway targets the insulin-like peptide Dilp6 to inhibit growth in Drosophila. Cell Reports. 28 (6): 1439–1446.
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S.W. Roth, M.D. Bitterman, Y.J. Lee, M.J. Birnbaum and M.L. Bland. 2018. Innate immune signaling in the Drosophila fat body blocks DILP signaling by uncoupling PI(3,4,5)P3 production and Akt activation. Cell Reports. 22 (10): 2550-2556.
Bland, M.L. 2016. Measurement of Carbon Dioxide Production from Radiolabeled Substrates in Drosophila melanogaster. Journal of Visual Experiments. Jun 27;(112). doi: 10.3791/54045.
Bland, M.L. and M.J. Birnbaum. 2011. Cell Biology. AdaPting to Energetic Stress. Science. 332 (6036): 1387-1388.
Bland, M.L., R.J. Lee, J.M. Magallanes, J.K. Foskett and M.J. Birnbaum. 2010. AMPK promotes growth in Drosophila melanogaster by regulating nutrient uptake and visceral muscle function in the gut. Developmental Biology. 344 (1): 293-303.
DiAngelo, J.R., M.L. Bland, S. Bambina, S. Cherry and M.J. Birnbaum. 2009. The immune response attenuates growth and nutrient storage in Drosophila melanogaster by reducing insulin signaling. Proc. Natl. Acad. Sci. 306 (49): 20853-20858.
Bland, M.L., R.C. Fowkes, and H.A. Ingraham. 2004. Differential requirements for SF-1 gene dosage in adrenal organogenesis versus adult endocrine function. Molecular Endocrinology. 18: 941-952.
Bland, M.L., C.A.M. Jamieson, S.F. Akana, S. R. Bornstein, G. Eisenhofer, M.F. Dallman, and H.A. Ingraham. 2000. Haploinsufficiency of steroidogenic factor-1 in mice disrupts adrenal development leading to an impaired stress response.
Proc. Natl. Acad. Sci. 97 (26): 14488-14493.